๐Ÿ—‚ ็ธฝ็›ฎ้Œ„ ๏ฝœ ๐Ÿ“– ่‹ฑๆ–‡ๅŽŸๆ–‡๏ผˆๆœฌ็ฏ‡๏ผ‰ ๏ฝœ ๐Ÿ“ ๅฎŒๆ•ด็ฟป่ญฏ ๏ฝœ โญ ็ฒพ่ฏ็ญ†่จ˜

Introduction

Gil Yosipovitch and Brian Kim Cutaneous Neurophysiology

Key features

ย„At least three subsets of pruriceptive C neurons exist that respond to histamine or other pruritogens and then activate distinct spinothalamic and spinoparabrachial tract neurons

ย„Peripheral itch mediators include histamine, proteases, and multiple interleukins

ย„Central itch mediators include opioids, gastrin-releasing peptide, and B-type natriuretic peptide

ย„There is an overlap between chronic itch and chronic pain, including activation of multiple brain areas and associated neuromediators and receptors, e.g. nerve growth factor, neurotrophin 4, transient receptor potential (TRP) channels

ย„Cross-talk between cutaneous nerve fibers and the stratum corneum is a possible mechanism for the pruritus associated with impaired barrier function (e.g. xerosis, atopic dermatitis)

ย„Therapies with topical and systemic drugs that reduce itch sensi- tization by counteracting the responsible mediators represent promising treatment strategies