๐Ÿ—‚ ็ธฝ็›ฎ้Œ„ ๏ฝœ ๐Ÿ“– ่‹ฑๆ–‡ๅŽŸๆ–‡๏ผˆๆœฌ็ฏ‡๏ผ‰ ๏ฝœ ๐Ÿ“ ๅฎŒๆ•ด็ฟป่ญฏ ๏ฝœ โญ ็ฒพ่ฏ็ญ†่จ˜

Introduction

Ariela Hafner and Eli Sprecher Ulcers

Chapter Contents

Synonyms:๏‚ก Ulcer โ€“ wound, sore ๏‚ก Venous ulcer โ€“ ulcer due to venous insufficiency/venous hypertension ๏‚ก Arterial ulcer โ€“ ulcer due to atherosclerosis ๏‚ก Neuropathic ulcer โ€“ mal perforans ๏‚ก Livedoid vasculopathyย โ€“ atrophie blanche, livedoid vasculitis ๏‚ก Pressure ulcer โ€“ decubitus ulcer, pressure sore, bed sore

Key features

ย„The history should include onset, course, symptoms, exacerbating and alleviating factors, past medical history, family history, social history, travel, and medications

ย„Important aspects of the physical examination include location, size, shape and depth of the ulcer, characteristics of the ulcer edge and base, and associated physical findings

ย„Helpful laboratory evaluation includes vascular studies, blood tests

(e.g. for thrombophilia), microbial cultures, and biopsy

ย„A biopsy of non-healing chronic ulcers is indicated to exclude carcinoma or other underlying diseases

ย„In addition to local care, therapy of ulcers should aim to correct or treat the underlying cause:

Venous insufficiency: compression

Arterial insufficiency: revascularization

Neuropathic or pressure ulcer: relief of pressure or other local factor that might be causing or exacerbating the ulceration

Infection: antibiotics

Neoplasm: surgery, chemotherapy, radiotherapy

Vasculopathy: treat the underlying cause of the occlusive disorder

Vasculitis or other inflammatory process (e.g. pyoderma gangrenosum): corticosteroids, immunosuppressive agents

ย„ A key factor in therapy is the promotion of wound healing:

Debridement: mechanical, enzymatic, autolytic

Dressings: maintain a moist wound environment

Infection control: antibiotics

Skin grafting or skin substitutes

Growth factors